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Ultimately, addressing MASH and T2DM as interconnected syndromes rather than isolated conditions is critical for developing next-generation, stage-specific, and patient-centered therapies

a , b , Comparison of wild-type GLP-1R and the R176 2.46b Q mutant by GLP-1 binding assay ( a ) and functional cAMP accumulation assay ( b )

Based on a systematic synthesis of available findings, we conclude that there is considerable support for the neuromodulatory potential of gut-based circulating peptides on reward responses

Outcomes and genotype-phenotype correlations in 52 individuals with VLCAD deficiency diagnosed by NBS and enrolled in the IBEM-IS database

Induction of NF-B by the Akt/PKB kinase

Kirstein, M., Aston, C., Hintz, R