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BPC-157 may accelerate blood vessel formation at injury sites through modulation of vascular endothelial growth factor (VEGF), supporting tissue oxygenation and nutrient delivery [1]

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Inhibition of autophagy in cells under high-fat conditions aggravates atherosclerosis, and iron accumulation often occurs in plaques, which may be caused by dysregulated mitophagy and the abrogation of the fragile metabolic balance in foam cells [225, 226]

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