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Not reduce it erase it entirely

If it holds up in larger, biopsy-confirmed studies, hepatic steatosis could become one of the areas where triple agonism has the strongest differentiated rationale though, as with everything about this molecule, that remains to be confirmed in the full program

Second, you must be seeing therapeutic effects like appetite suppression and weight changes

Malkowski notes observations of individuals on GLP1 medications demonstrating gut microbiome dysbiosis, reduced diversity, reduced phyla abundance, and key bacteria

This in turn leads to excessive storage of underutilized glucose in the liver (glycogenesis), the potential for developing fatty liver (ie, nonalcoholic steatohepatitis), and conversion of excess carbohydrates to fatty acids and triglycerides, with subsequent storage in adipose tissue