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In vivo investigations corroborated the in vitro results, demonstrating that Au@miR-183i NCs effectively targeted tumors, induced robust ICD, and significantly enhanced PD-L1 immunotherapy, finally eliciting a vigorous systemic anticancer immune response in mice models with HCC [317]

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Blood flow can also trigger NO production through eNOS phosphorylation via protein kinase B, without needing more calcium [1]

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