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Acetyl-L-carnitine deficiency as a cause of altered nerve myo-inositol content, Na,K- ATPase activity, and motor conduction velocity in the streptozotocin-diabetic rat Metabolism 1996

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The simultaneous interaction with the endogenous Ub-E2-E3 complex and the protein of interest results in the generation of an Ub-E2-E3-neosubstrate complex, ultimately leading to ubiquitinylation and degradation of target protein