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It has been observed that the apoptotic process in paclitaxel induced-painful neuropathy is due to the permeability transition pore opening, with the consequent cytochrome c release and Ca 2+ homeostasis dysregulation (Areti et al., 2015) reported that the selective pharmacological modulation of the mitochondrial electron transport chain (ETC) (at level of complex I or III) in vivo could also reverse or attenuate the above-mentioned symptoms, but with deleterious effects on motor coordination in the case of complex I inhibition at 3 and 24 h after paclitaxel administration

Strengthens the Immune System By promoting healthy red and white blood cell production, B12 enhances the immune system's ability to fight infections and maintain overall health

Impaired incretin effect is an early sign of glucose dysmetabolism in nondiabetic patients with psoriasis

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The intronic variant rs56159231 may influence ovarian cancer risk by disrupting Er and TCF11::MafG binding sites, modulating PLEKHM1 -mediated autophagy