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Penicillamine and trientine, both copper chelators, will bind and inactivate the copper component of GHK-Cu, likely eliminating its therapeutic effect [10]

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83 In collagen VI-related dystrophies, reduced BECN1 hampers autophagy initiation and myofiber integrity, whereas autophagy restoration rescues the phenotype

This reaction simultaneously depletes two critically important metabolic cofactors: SAM (the universal methyl donor for hundreds of epigenetic and biosynthetic reactions) and nicotinamide (the precursor for NAD+ biosynthesis via the salvage pathway)

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Patel RT, Gay JJ, Fagan KK, Eikenberg JD