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However, another report demonstrated mitochondrial stress and bioenergetics defects in GPx1 null mice ( Figure 3 ), and hence plays a key role in protecting phospholipids, cholesteryl esters and cardiolipin and defense against apoptosis and maintenance of ETC and OXPHOS ( + / - cells are sensitive to oxidative stress triggers (Muller et al., 2007)

Genomic Responses in Mouse Models Poorly Mimic Human Inflammatory Diseases

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